98%+ Purity

MOTS-c

Mitochondrial-derived peptide

CAS Number

1627580-64-6

Molecular Weight

2174.64 g/mol

Available Forms
Lyophilized Powder
Available Sizes
5mg10mg20mg

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Certificate of Analysis

Every batch tested with COA provided

98%+ Purity

HPLC verified purity standards

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Research Areas
Mitochondrial BiologyExercise PhysiologyMetabolic ResearchAging Studies

Mitochondrial-derived peptide for metabolic and longevity research. MOTS-c is a 16-amino acid peptide encoded in the mitochondrial genome with demonstrated effects on glucose metabolism and exercise mimetic properties.

MOTS-c Research Compound

Research Use Only

This product is intended for laboratory research purposes only. It is not intended for human or veterinary use, food, cosmetic, household, or agricultural applications. Not for human consumption.

Overview

MOTS-c (Mitochondrial Open Reading Frame of the 12S rRNA-c) is a 16-amino acid peptide encoded within the mitochondrial genome (12S rRNA gene). Discovered in 2015 by Dr. Pinchas Cohen's laboratory at USC, MOTS-c represents a new class of mitochondrial-derived peptides (MDPs) that act as signaling molecules. It has been characterized as an "exercise mimetic" due to its effects on glucose metabolism and AMPK activation.

Molecular Characteristics

PropertyValue
Molecular FormulaC102H152N28O25S2
Molecular Weight2174.64 g/mol
CAS Number1627580-64-6
SequenceMRWQEMGYIFYPRKLR
Amino Acids16
Encoded ByMitochondrial 12S rRNA
Purity≥98% (HPLC)

Mechanism of Action

MOTS-c exerts metabolic effects through several pathways:

AMPK Activation

  • Direct AMPK phosphorylation
  • Energy sensor activation
  • Glucose uptake stimulation
  • Fatty acid oxidation promotion

Folate-Methionine Cycle

  • AICAR accumulation
  • De novo purine synthesis inhibition
  • Methionine cycle interaction
  • One-carbon metabolism modulation

Nuclear Translocation

  • Stress-induced nuclear entry
  • Gene expression regulation
  • Antioxidant response elements
  • Adaptive response coordination

Mitochondrial Function

  • mtDNA-encoded signaling
  • Mitochondrial-nuclear communication
  • Cellular stress response
  • Energy homeostasis

Research Applications

Exercise Physiology

  • Exercise mimetic studies
  • Physical performance assessment
  • Skeletal muscle adaptation
  • Endurance capacity research

Metabolic Research

  • Glucose homeostasis studies
  • Insulin sensitivity assessment
  • Obesity model research
  • Metabolic syndrome interventions

Aging Studies

  • Age-related MOTS-c decline
  • Healthspan intervention research
  • Mitochondrial aging
  • Longevity pathway studies

Stress Response

  • Metabolic stress adaptation
  • Cellular resilience research
  • Mitohormesis studies
  • Environmental stress response

Available Formats

SizeFormatCatalog Code
5mgLyophilizedMOTS-5
10mgLyophilizedMOTS-10
20mgLyophilizedMOTS-20

Handling & Storage

  • Store lyophilized peptide at -20°C
  • Protect from light and moisture
  • Reconstitute with bacteriostatic water
  • Reconstituted solution stable at 2-8°C for up to 2 weeks
  • Contains methionine (oxidation-sensitive)
  • Use argon/nitrogen overlay for long-term storage

Quality Specifications

  • Purity: ≥98% by HPLC
  • Appearance: White lyophilized powder
  • Solubility: Soluble in water
  • Endotoxin: Less than 1 EU/mg
  • Certificate of Analysis provided

Key Research Findings

StudyFinding
Lee et al. (2015)MOTS-c regulates insulin sensitivity and metabolic homeostasis
Reynolds et al. (2021)MOTS-c levels decline with age; associated with physical activity
Kim et al. (2019)Nuclear translocation under stress

Regulatory Notes

As of 2024, MOTS-c was included in FDA's list of compounds not suitable for compounding. This applies to compounded medications, not research use.

References

  1. Lee C, et al. "The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance." Cell Metab. 2015.
  2. Reynolds JC, et al. "MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis." Nat Commun. 2021.
  3. Kim KH, et al. "MOTS-c nuclear entry regulates adaptive response to metabolic stress." Cell. 2019.

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